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Australian State/Territory : VIC
Research Topic : B-Lymphocytes
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  • Funded Activity

    The Role HLA-E In The Activation Of Decidual Natural Killer Cells

    Funder
    National Health and Medical Research Council
    Funding Amount
    $7,044.00
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    Funded Activity

    Characterisation Of Anti-HBs Responses In Patients Undergoing Functional Hepatitis B Cure: Implication For Future Therapies

    Funder
    National Health and Medical Research Council
    Funding Amount
    $723,649.00
    Summary
    The hepatitis B virus causes liver cirrhosis and liver cancer. There is no cure for hepatitis B. However, a small number of patients can naturally rid themselves of the virus. We have identified 14 of these individuals and discovered that they have a unique immune response that is responsible for these “natural” cures. We plan to characterise this immune response and turn it into a therapeutic vaccine which can be used to cure patients who are still chronically infected.
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    Funded Activity

    The Role Of Non-classical MHC Class I Molecules In Adaptive Immunity

    Funder
    National Health and Medical Research Council
    Funding Amount
    $443,834.00
    Summary
    Specialised proteins called MHC class Ia molecules (MHC-Ia) stimulate killer T cells to lyse virus infected cells. In contrast, the function of the closely related MHC-Ib is uncertain. Recent findings have demonstrated that MHC-Ib can also be recognised by T cells and this interaction is important in the control of viral infections. However, despite the similarity to MHC-Ia, it is unclear how this interaction occurs. This project aims to investigate how killer T cells recognise MHC-Ib molecules.
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    Funded Activity

    Investigating The Host Determinants Of Viral Clearance Versus Collateral Pathology In Chronic Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $1,250,756.00
    Summary
    Hepatitis B virus has infected over 2 billion people. Some people control the virus but it remains incurable and there is a lifelong risk of liver cancer. Understanding how host cells interact with the virus, the mechanisms the cells use in an attempt to eliminate the virus and the mechanisms the virus uses to sabotage these responses, will provide insights that could lead to therapies. Potential therapies could be applicable to other infections like HIV-1 and tuberculosis.
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    Funded Activity

    Determinants Of Sustained Virological Response After Discontinuation Of Long-term Nucleoside Analogue Therapy In Chronic Hepatitis B Patients

    Funder
    National Health and Medical Research Council
    Funding Amount
    $976,778.00
    Summary
    Guidelines currently recommend lifelong treatment for patients with chronic hepatitis B, with associated cost and risks of drug resistance and side effects. It has recently been suggested that up to 50% of patients may safely and successfully stop drug after long-term treatment. Our project will identify which patients can safely stop treatment, by performing detailed studies of the human immune system and the hepatitis B virus. This will be an important advance for patient care.
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    Funded Activity

    How Do Cross-reactive Memory B Cells Affect Influenza Vaccine Titers?

    Funder
    National Health and Medical Research Council
    Funding Amount
    $798,049.00
    Summary
    Influenza vaccines are updated frequently to protect against the highly variable influenza virus. Despite careful selection of vaccine viruses, most influenza vaccines provide only modest protection and protection is poor some years. In turn, the response to vaccination varies between individuals. This probably reflects complex and variable histories of influenza infection and vaccination. The project investigates how past influenza exposure influences vaccine responses and effectiveness.
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    Funded Activity

    The Role Of Co-signalling Receptors In Cytotoxic Lymphocyte Activity During Infection And Cancer

    Funder
    National Health and Medical Research Council
    Funding Amount
    $739,657.00
    Summary
    Cytotoxic lymphocytes (CLs) are immune cells that detect and kill cancer cells. CLs recognise ‘stress’ proteins on cancer cells through specialised receptors, and this provides the signal for them to kill. However, some cancer cells, such as leukemic cells, can interfere with this recognition to avoid killing by immune cells. This project will investigate the mechanism of recognition and killing of cancer cells by CLs, using both mouse models and cells from patients with acute myeloid leukemia.
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    Funded Activity

    The Role Of IL-18 In Proliferative And Crescentic Glomerulonephritis

    Funder
    National Health and Medical Research Council
    Funding Amount
    $56,177.00
    Summary
    Inflammation of the small filters with the kidneys, known as glomerulonephritis, is the commonest cause of kidney failure in Australia. People whose kidneys have failed need either kidney dialysis or a kidney transplant. Our understanding of the immune events that cause glomerulonephritis is patchy. However, it is known that T cells are the directors of immune responses in the body and direct the immune response in glomerulonephritis. Chemical messengers known as cytokines direct the way T cells .... Inflammation of the small filters with the kidneys, known as glomerulonephritis, is the commonest cause of kidney failure in Australia. People whose kidneys have failed need either kidney dialysis or a kidney transplant. Our understanding of the immune events that cause glomerulonephritis is patchy. However, it is known that T cells are the directors of immune responses in the body and direct the immune response in glomerulonephritis. Chemical messengers known as cytokines direct the way T cells behave. One of these cytokines, known as interleukin-18, has been shown to stimulate T cells and other immune cells to induce inflammation that is helpful when the body is fighting infection but is harmful in immune diseases. This project will determine the role of interleukin-18 in glomerulonephritis by studying the way it talks to T cells and the mechanisms by which it incites inflammation in the kidney. Mice with glomerulonephritis will be treated by blocking the actions of interleukin-18 to discover whether interleukin-18 produced by the animal is important in kidney damage induced by glomerulonephritis, to understand the way in which this cytokine works and to assess whether blocking interleukin-18 could be a useful treatment for glomerulonephritis in humans. Current treatments for glomerulonephritis are often ineffective and have unwanted side effects. Knowledge of the way interleukin-18 participates in the immune response in glomerulonephritis may lead directly or indirectly to more effective and more targeted treatments for different forms of glomerulonephritis.
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    Funded Activity

    New Drug Combinations To Enhance Elimination Of Hepatitis B Infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $888,304.00
    Summary
    We have developed a therapy that kills hepatitis B virus infected cells and promotes elimination of infection. We are now testing novel drugs that can be used to maximise the efficacy of our new treatment to promote better outcomes that may be translated to other infections.
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    Funded Activity

    Long Term Persistence Of HIV In The Liver And The Clinical Impact On HIV-HBV Co-infection

    Funder
    National Health and Medical Research Council
    Funding Amount
    $1,393,245.00
    Summary
    This grant will address a major question in HIV cure research - the role of the liver as an HIV reservoir and the impact of HIV persistence in HIV-infected patients on suppressive antiretroviral therapy (ART) on liver disease, in the setting of HIV-HBV co-infection. We will trial a novel intervention to reduce HIV infection of the liver that could potentially reduce chronic liver disease in this setting.
    More information

    Showing 1-10 of 11 Funded Activites

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